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DHEA mechanism as antidepressant may act via modulation of the GABAARs activity. The GABA ergic system can mediate depression (Lloyd et al., 1989). In depressed patients, cerebral spinal fluid numbers of GABA are a lesser amount than in healthy controls (Gerner and Hare, 1981; Gold et al., 1980). Furthermore, low GABA content is correlated with depression in post mortem temporal and frontal cortices from patients with Alzheimer’s disease (Garcia Alloza et alii, 2006). During these patients, a high density of GABAARs correlates while using harshness of depression at www.07runescapegolds.com.au.
Inspite of the increased cortical concentration of GABA upon antidepressant treatment of healthy individuals (Bhagwagar et al, 2004) and electroconvulsive treatments for depressed patients (Sanacora et aliae, 2003), anti depressants that affect GABA have yet to be developed. The subunit composition determines the pharmacological properties of GABAAR. Drugs, including neurosteroids, which communicate with the receptors, make a difference to the subunit mRNA expression (Sun et al, 2004). GABAAR usually contains two two and only a or with all the being rare (Sun et alibi, 2004). GABAARs containing a fairly than a are more sensitive to neurosteroids (Belelli et alibi, 2002), and mice lacking GABAA are less responsive to neurosteroids (Brown et alibi, 2002; Mihalek et alibi, 1999, 2001; Stell et alibi, 2003; Wohlfarth et al, 2002). This might account for the abnormal behavior of patients with 1q36 deletion, including the human being GABAA gene (Windpassinger et al, 2002).